Clinical and molecular characterization of a second case of 7p22.1 microduplication

  • Egle Preiksaitiene
  • , Jurate Kasnauskiene
  • , Zivile Ciuladaite
  • , Birute Tumiene
  • , Philippos C. Patsalis
  • , Vaidutis Kučinskas

Research output: Contribution to journalArticlepeer-review

Abstract

The use of high-resolution microarray technology for investigation of patients with intellectual disability and/or congenital anomalies provided the unique possibility to identify new microdeletion/microduplication syndromes and discover the dosage sensitive genes, which are implicated in the manifestation of various genetic conditions. Microduplication of the 7p22.1 region, 1.7Mb in size, has very recently been reported, representing the smallest interstitional 7p duplication, associated with specific facial features and speech delay. We report on a patient with an even smaller 7p22.1 de novo microduplication, 1Mb in size, detected in a 14.5-year-old patient with mild intellectual disability and similar facial dysmorphism, including macrocephaly, ocular hypertelorism, low-set ears, and other features. There are 15 RefSeq genes included in this duplication. ACTB gene is a strong candidate gene for the alteration of craniofacial development. Further cases with similar duplications will contribute to the delineation of a potential new microduplication syndrome of 7p22.1.

Original languageEnglish
Pages (from-to)1200-1203
Number of pages4
JournalAmerican Journal of Medical Genetics, Part A
Volume158 A
Issue number5
DOIs
Publication statusPublished - May 2012
Externally publishedYes

Keywords

  • β-actin
  • 7p22.1 microduplication syndrome
  • ACTB gene
  • Craniofacial abnormalities
  • Developmental delay
  • Skeletal abnormalities

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