Genetic background modifies amyloidosis in a mouse model of ATTR neuropathy

Keyphrases

• Mouse Model 100%
• Amyloidosis 100%
• Neuropathy 100%
• Genetic Background 100%
• V30M 66%
• Amyloid Deposition 66%
• Penetrance 66%
• Age of Onset 33%
• Complement Activation 33%
• Transthyretin 33%
• World Population 33%
• Megalin 33%
• Disease Marker 33%
• Immunohistochemistry 33%
• Apoptosis Activation 33%
• Transthyretin Amyloidosis 33%
• Endoplasmic Reticulum Stress (ER stress) 33%
• Two-line 33%
• Clusterin 33%
• Ubiquitin-proteasome System 33%
• Transgenic Mice 33%
• Environmental Modifiers 33%
• Genetic Modifiers 33%

Pharmacology, Toxicology and Pharmaceutical Science

• Amyloid Protein 100%

Biochemistry, Genetics and Molecular Biology

• Amyloid Protein 66%